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Curr Pharm Des 20(18):30833092

doi: 10.1002/advs.202512828 17

On one hand, NADH is overproduced due to overnutrition and hyperglycemic activation of the polyol pathway [81, 103]

Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]

[The Working Group noted that the urinary excretion data were consistent with the conversion of methanol to formate, with the rate of formate production not exceeding the rate of formate excretion