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For instance, studies have shown higher NK presence in tumors to be associated with lower pyruvate to lactate conversion, an effect hypothesized to be mediated by overexpression of FOXD1 via USP21

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However, when ER stress is persistent and severe, UPR is insufficient to restore ER function, it promotes cell apoptosis by upregulating the expression of GADD153/CHOP, by activating caspase-12, and by inhibiting Bcl-2 expression

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