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One possible explanation is that, despite encoding the GGLG and YF motifs, the susceptibility to hyperoxidation for each of these 10 species may in fact be sufficiently low that AhpC-PRX1 inactivation does not occur

However, biochemists speculate that there may have been some concurrent advantage of this mutation that caused it to persist and spread in the human lineage

Manta et al., 2019

Verdict: Both are excellent

This expanded understanding of myelin and its relationship with neurons sheds light not only on its role in normal physiological functions but also in the pathology of various neurological and psychiatric disorders