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Although the exact pathogenic mechanism of DKD is still unclear, programmed cell death has been demonstrated to participate in the occurrence and development of diabetic kidney injury, including ferroptosis

These advances, combined with the increase in using cellular therapies such as exosome-based therapies, promise not only to mitigate symptoms but literally reconstruct the very infrastructure of damaged brain tissue

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Abstract Glutaredoxins are key players in cellular redox homoeostasis and exert a variety of essential functions ranging from glutathione-dependent catalysis to iron metabolism