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The c-MET serves as an attractive therapeutic target in SCLC, as shown through small interfering RNA (siRNA) and selective prototype c-MET inhibitor SU11274, inhibiting the phosphorylation of c-MET itself and its downstream molecules such as AKT, S6 kinase, and ERK1/2

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The top candidates positively affecting steady-state levels of pAkt were abundance of Akt, abundance of PI3K, and abundance of GAB1, while PI3K inactivation, pAkt deactivation, and Gab1 pMet dissociation were the top parameters negatively affecting the steady-state levels of pAkt (Fig

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