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Fatty acid conjugation as used in Semaglutide and Retatrutide promotes reversible albumin binding that shields the peptide from enzymatic degradation and slows renal filtration, extending the half-life from minutes to days

SLC39A1 interacted with DRP1 to impair mitochondrial quality control, promote autophagy and reduce MMP and ROS production, thereby promoting cell proliferation and inhibiting apoptosis

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ATRA also triggers kinase cascades, including the p38, MAPK, and ERK pathways, that are crucial for alveolar epithelial repair, immune cell signaling, and maintaining a balanced response to oxidative stress [29,30]