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Neuroprotective Agents for Clinical Trials: National Institute of Neurological Disorders and Stroke

A new APE1/Ref-1-dependent pathway leading to reduction of NF-kappaB and AP-1, and activation of their DNA-binding activity

Dorostkar et al., 2015), and correlates with reduced glutamate receptor densities, synaptic depression, and memory deficits (Hsieh et al., 2006

10.1016/j.psychres.2018.03.061 106 MoussaC.HebronM.HuangX.AhnJ.RissmanR

By decreasing production of pro-inflammatory cytokines including IL-6 and TNF-alpha, BPC-157 limits ongoing tissue destruction while simultaneously promoting repair