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Loss of sirt1 promotes prostatic intraepithelial neoplasia, reduces mitophagy, and delays park2 translocation to mitochondria

Increasing efforts have been endeavored to identify potential inhibitors against gasdermins, which are key executors in pyroptosis and are expected to be promising therapeutic targets for diverse diseases including but not limited to septic shock, cancer, and tissue damage

More efforts are necessary to establish the therapeutic window for cancer without contributing to other NAD-derived illness or toxicity

Additionally, the differential effects of exercise intensity and training load on ferroptosis pathways remain poorly understood, representing a critical research gap