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10.1002/jpen.1744 295 LubomskiM.DavisR

459,460 The anti-inflammatory effects of GLP-1RAs contribute to the attenuation of atherosclerotic plaque lesion development through various mechanisms

Indeed, the combination of CB-839 with ATO or HHT exacerbates mitochondrial oxidation and apoptosis in the cell lines, mouse models, and patients of AML [11]