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Notwithstanding these advances, several critical barriers limit translational potential

Additionally, cell death and membrane damage cause an additional massive release of glutamate and ATP, aggravating the initial ischemia-induced damage (Matute and Ransom, 2012)

Mechanistically, macrophages post-MI recognize DAMPs released from necrotic cells through Toll-like receptors (TLRs), activating downstream signaling pathways such as the mTOR pathway and entering an activated state with increased energy demands (224, 226)

This allows the perseverance of unhealthy habits and adherence to uniformly processed foods (120)

This examination involved three different stainings including Hematoxylin and eosin (H&E), Gmri trichrome, and Nicotinamide adenine dehydrogenase (NADH)-TR