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For example, when investigating the hypothesis that gap junctions transport toxic metabolites to neighboring cells and support their killing during APAP and thioacetamide hepatotoxicity, the fact that the new gap junction inhibitor 2-aminoethoxydipenyl-borate (2APB) protected against the injury appeared to support the hypothesis [154]

10.1111/j.1567-1364.2012.00834.x FEMS Yeast Res

38 , e382923 (2023)

Goss AM, Chandler-Laney PC, Ovalle F, Goree LL, Azziz R, Desmond RA, Bates GW, Gower BA

The generation of ferroptosis signals requires the formation of CoA derivatives of PUFAs and their binding to phospholipids, which represents a potential target for treating diseases associated with ferroptosis