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At the end of follow-up, a higher prescription of SGLT2i was seen (20% vs 6%, p 10% from baseline

TETA, typically administered as a dihydrochloride salt, restores cardiac function by rectifying copper homeostasis and repairing mitochondrial integrity

Melatonin can also influence mitochondria-dependent apoptotic processes by interfering with the release of ROS, cytochrome c, and cell death regulatory proteins [47]

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