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This evidence suggests that acute and repeated injuries have distinct temporal patterns of glucose utilization, neuroinflammatory responses, and tau hyperphosphorylation

These alterations are usually manifested by an activation and elevated concentrations of pro-inflammatory cytokines (IL-1, TNF, IL-2, IL-6, IL-17), interferon gamma (INF-), and C-reactive protein (CRP) as well as a decrease in IL-4 levels, disrupting normal immune cell balance ( Inflammatory processes are closely connected to mitochondria

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J Immunol 199(8):29762984

Autonomic dysfunction in long COVID: rationale, physiology and management strategies