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Description

Dysregulated calcium fluxes promote malignant progression by disrupting cell cycle regulation, inhibiting apoptosis, and facilitating epithelial-mesenchymal transition (EMT) and cytoskeletal remodeling [23-27]

Briefly, circulating neutrophils patrol the blood continuously, until they encounter pathogen-derived chemoattractants and inflammatory signals released by immune cells and non-hematopoietic epithelial and endothelial cells in response to tissue injury or infection

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