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Axonal plasticity and functional recovery after spinal cord injury in mice deficient in both glial fibrillary acidic protein and vimentin genes

In contrast, most other approved therapies may reduce oxidative damage only indirectly by attenuating inflammation, but they are not specifically designed to target persistent reactive oxygen and nitrogen species production, mitochondrial dysfunction, iron-associated oxidative injury, or chronic microglial activation

This structure plays a vital role in maintaining the structural integrity of the BBB

Thus, a tight coupling was demonstrated between neuronal activation and glucose utilization in astrocytes (Magistretti, 2006), an intrinsic feature of astrocytes not specifically linked to culture conditions or cell origin (Pellerin et al., 2007)