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J Neurosurg:1-8

A key adaptive response is observed upon DNMT3A loss, in which compensatory hypertrophic signaling can restore metabolic and contractile function, demonstrating that its regulatory role is context-dependent and can be bypassed under stress

Schaffalitzky De Muckadell, O

This highlights the ongoing need for accessible, non-invasive biomarkers that can aid in the timely detection, accurate diagnosis, and monitoring of disease progression and future treatment response

Mitochondrial dysfunction plays a pivotal role in PD-associated oxidative pathology through four interconnected mechanisms: progressive depletion of nigral glutathione (GSH