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125 The expression and activity of the SOP enzymes appear to be strongly related to the H 2 S levels to which the corresponding cells or tissues are exposed

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Whereas CHAC1 overexpression promoted GSH depletion, ROS accumulation and promoted drug-induced apoptosis in resistant cells (Supplementary Fig

Elevated concentrations of H 2 S damage pulmonary epithelial cells, promote inflammatory-cell infiltration, increase cytokine release, and culminate in lung injury ( 2 S have a protective effect on the lungs

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