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Abstract Background Androgen receptor (AR) ligand-binding domain (LBD) mutations occur in ~20% of all castration-resistant prostate cancer (CRPC) patients

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Although viruses are extremely minuscule, they are flexible and promote tumorigenesis through different pathways involving signal transduction changes, the DNA damage response and immune regulation (Krump and You, 2018)

Eleven studies contained some concerns about the risk of bias

Androgen receptor defects: historical, clinical, and molecular perspectives