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The microglial component of amyotrophic lateral sclerosis

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Neutrophils induce paracrine telomere dysfunction and senescence in ROS-dependent manner

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Pyridoxine hydrochloride (vitamin B6) I.P

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For open access purposes, the author has applied a CC BY public copyright license to any author accepted manuscript version arising from this submission

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Because cellular iron output is mediated by FPN1 and polycupric ferrous oxide enzymes (such as ceruloplasmin), a decrease in the expression of FPN1 or ceruloplasmin can increase the sensitivity of cells to ferroptosis [29,30,31,32]

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