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Hemorrhagic transformation occurs through a disruption in tight junction proteins, and investigations into this underlying mechanism found that dysregulated extracellular proteolysis in the vascular matrix after tPA contributes to hemorrhagic transformation (Chandler et al., 1990

Also influences stability, though less dramatically than temperature or light

Research demonstrates effects across wound healing, tendon regeneration, gastroprotection, and angiogenesis models

Food & Drug Administration, et al

In the context of cancer, multiple clinical trials using CQ or HCQ either in combination or as monotherapy are currently in progress showing partly promising results in terms of anticancer effects and toxicity in early phase studies