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YAP inhibits insulin-like growth factor (IGF) during the IGF pathway activation, which leads to the stabilization and nuclear translocation of -catenin (promoting heart development) (Xin et al., 2011)

It remains unclear whether distinct death signals are activated sequentially, in parallel, or undergo context-dependent modulation at the single-cell or population level-particularly across different respiratory epithelial subtypes and immune cell populations under diverse stress conditions

Fosphenytoin can be used with an intramuscular administration route

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It may be genetic differences or overall toxic body burden that leads to differences in susceptibility