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Acyl-CoA synthetases are the gateway to mitochondrial or peroxisomal -oxidation, or synthesis of triacylglycerols, phospholipids and other acylated products, while the CoA esters and acylated proteins are also acting in cell signaling [33]

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10.1016/j.phymed.2022.154289 62 LiuW.ShangJ.DengY.HanX.ChenY.WangS.et al (2022b)

Tao JX, Zhou WC, Zhu XG

Experiments have shown that female mice lacking Ambra-1 displayed autism-like behavior, supporting the findings of inhibited autophagy in ASD [286]