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Within the intestinal epithelium, S1P serves as a crucial regulator of epithelial cell barrier function by activating the receptor S1PR1 located in intestinal blood vessels

Remarkably, reactive oxygen species (ROS) generated through colonic urea metabolic processes demonstrate capacity to induce renal tubular epithelial cell mitochondrial functional catastrophe, exponentially accelerating tubulointerstitial fibrotic transformation and establishing a devastating gut-derived urearenal injury pathophysiological cascade amplification phenomenon (Rukavina Mikusic et al., 2020

9E) indicated dominant branching of Pseudomonadota and minimal representation of Bacteroidota in the VPA-ASD group, which was attenuated by hCiPSC treatment