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These studies cannot definitively establish the presence or absence of risk due to several methodological limitations

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[127] studied MT-2A expression in visceral adipose and subcutaneous tissues of people with obesity with and without diabetes, and they showed that MT-2A might be involved in the modulation of insulin resistance in fat cells

Mitochondrial dysfunction enhances the production of ROS, which serve as critical regulators of cell fate and central mediators of ferroptosis 102