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Herein, a putative co-existence of defective DNA damage repair mechanism, together with the therapy-specific DNA damage, may explain the role of Taxol derivatives in t-SMD/t-AML that arise from cellular populations with an incredibly higher turnover (Smith, 2003)

Molecular docking predicts interactions with bacterial enzymes, while physicochemical modeling estimates permeability across ocular barriers (Singh and Shukla, 2012)

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Available from: Desai B, Assid E, Jacobs G, Dasgupta A, Williams G, Choate WS, et al

Geographical distribution of MTHFR C677T, A1298C and MTRR A66G gene polymorphisms in China: findings from 15357 adults of Han nationality