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These include ~50 gene loci, with mutations in three Mendelian genes myocilin (MYOC), optineurin (OPTN) and TANK binding kinase (TBK1) accounting for ~5% of POAG cases with little influence from other genes or environmental factors (12, 20)

The first one is hyperglutamate theory, which accepts that glutamate levels are high in the serum and plasma of individuals with ASD, and the second one is the hypoglutamate theory, which states that there is dysfunction in glutamate receptors in ASD and glutamatergic agonists may have a positive effect on some of ASD symptoms [55]

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doi: 10.1016/j.rbmo.2017.02.006 113 TremellenK

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