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These effects are attributable to redox regulation by SFN, since (i) SFN induced ROS and in parallel GSH depletion in primary human T-cells and (ii) thiol-containing antioxidants abolished the immunosuppressive effect of SFN

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Consistently, AFAP-AS1 knockdown inhibited tumorigenesis in vivo

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The imaging of changes in intracellular ROS revealed that elastin treatment increased the generation of ROS in elastin-treated A549 cells