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Isoniazid (INH), as an anti-tuberculosis drug, damages the liver and induces lipid peroxidation leading to apoptosis of liver cells through glutathione depletion (175)

Regarding relapsed drug-resistant LSCs, combined therapy is necessary due to the different mechanisms of drug resistance

low GSH-producing cells)

Beyond its reproductive toxicity, it significantly impacts human health by disturbing intracellular calcium homeostasis, generating oxidative stress, and interfering with critical cell signaling pathways, ultimately leading to cellular dysfunction (49)

Nerve repair is the slowest, with meaningful recovery ranging from 8 to 16 weeks, depending on the extent