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Of note, pharmacologically induced PFC NMDAR hypofunction leads to increased oscillations [99] while genetic deletion of NMDAR1 subunits in pyramidal neurons increases oscillations, cell excitability and sEPSC frequency [100], similar to the present data

In addition, amino acid starvation upregulates SLC38A2 via activation of GCN2 and ATF4, which help to maintain the intracellular glutamine pool 36,90

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POPs increase your bodys need for glutathione, which means levels need to be higher the more you are exposed to these pollutants

The success of autophagy-modulating therapies will rely not only on their mechanistic efficacy in preclinical models but also on their capacity to achieve sustained, tolerable, and reproducible effects in heterogeneous patient populations