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The proinflammatory immune response with high levels of inflammatory processes which is present in most infected cases is associated with hypercoagulable state [10]

The mechanism of CYP is a complex cascade of individual steps involving the interaction of protein redox partners and consumption of reducing equivalents, usually in the form of NADPH

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Ferroptosis: mechanisms, biology, and role in disease

To address this knowledge gap, here we develop a mathematical model that incorporates the most essential molecular elements for NADPH production, ROS generation, GSH synthesis, and detoxification of ROS by GSH in the outer retina, formed by photoreceptors and the RPE (see Fig