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This review aims to systematically elucidate the core molecular mechanisms of ferroptosis and cuproptosis, explore their specific regulatory networks and resulting metabolic vulnerabilities in different GI tumors, comprehensively summarize therapeutic intervention strategies targeting these pathways, and discuss the prospects and challenges of their clinical translation, to provide new insights for overcoming refractory GI tumors

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H 2 S is oxidized by SQR to generate sulfane sulfur (S 0 ) which has six valence electrons and no charge forming persulfide (SQRS-S)

Para favorecen la colonizacin de la microbiota intestinal al igual que ocurre en nios alimentados con leche materna, ciertas formulas de continuacin contienen probiticos y prebiticos