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[DOI] [PMC free article] [PubMed] [Google Scholar] 177

Public Health 18 , 1415 (2021)

In our study, an earlier and greater decline of T-AOC was found in the hippocampal of APP/PS1 mice compared to WT mice, indicating the exacerbated decline of antioxidant capacity in APP/PS1 mice

This mechanism is critical in acetaminophen overdose, where the toxic metabolite NAPQI depletes hepatic glutathione N-acetylcysteine administration replenishes GSH and prevents liver failure

Although antioxidant therapy is increasingly employed in the treatment of male infertility, there is still an absence of validated biomarkers and standardized clinical protocols to monitor redox balance and minimize the risk of RS-related adverse effects [14]