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Neuronal UCPs (UCP2, UCP4, BMCP1/UCP5) are integral membrane proteins located in the mitochondrial inner membrane, which positively affect neuronal function by preventing neuronal degeneration through regulating UCP-mediated proton transport and ROS production [13]

Although there is a broad clinical spectrum of glutathione synthetase deficiency, manifestations usually include hemolytic anemia, often include metabolic acidosis, and sometimes include progressive neurologic symptoms as well as other systemic signs (eg, respiratory distress, sepsis, hyperbilirubinemia) (62

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The proportion of each cell type in the heart showed no significant changes by PD-1 inhibitor treatment (Figure 2E)