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Part of the enzyme activity that was detected might be due to minimal microsomal contamination from this method

These results suggest that UgsL accelerates the degradation of OsSRF3 by enhancing its association with the ubiquitin-26S proteasome

Despite the overwhelming higher prevalence of SAD, 80% of AD preclinical research focuses on the use of transgenic mouse lines carrying mutations of FAD while only a few models use chemical induction, senescence, and cellular reprogramming from SAD patients [145]

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