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Elevated levels of these proinflammatory cytokines lead to the activation of the NF-B and MAPK signalling pathways [117]

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When iron load increases (e.g., elevated iron stores, high serum iron levels) [26], elevated hepcidin directly binds to membrane-bound FPN, promoting its internalization and degradation within intestinal epithelial cells, thereby inhibiting iron absorption into the bloodstream [27]

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