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As the primary organ responsible for steroid metabolism, the liver is particularly susceptible to the toxic effects of 17--alkylated oral anabolicandrogenic steroids, such as stanozolol and methyltestosterone ( Mechanistic studies suggest that anabolicandrogenic steroids impair mitochondrial -oxidation and deplete intracellular glutathione stores, resulting in lipid peroxidation and activation of apoptotic signalling pathways

[DOI] [PubMed] [Google Scholar] 72.Ju J., Li X.-M., Zhao X.-M., Li F.-H., Wang S.-C., Wang K., Li R.-F., Zhou L.-Y., Liang L., Wang Y., et al

This process can be antagonized by methylation inhibitors silencing DNMT3a and SEPT9 overexpression, thereby reducing liver fibrosis, including upregulation of apoptosis-related proteins in myofibroblasts, and reduction of TGF-1-induced expression of -SMA and COL1A1

Research indicates that levels are often highest during sleep, as the body shifts into "repair and detox" mode

To further simplify data analysis we implemented two essential features in our assay