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doi: 10.3390/arm92050039 32 XiongJZhouLTianJYangXLiYJinRet al

The RIPK1/RIPK3/MLKL complex induces necroptosis through multiple mechanisms, including promoting mitochondrial autophagy, producing mitochondrial reactive oxygen species, and altering mitochondrial permeability, leading to mitochondrial dysfunction (141)

Rogers, T

It is silenced early in development in normal somatic cells, with the exception of stem cells, and reactivated specifically in tumor cells via hotspot mutations in the TERT promoter (5,7)