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Recent elegant work has definitively identified that TP53 missense mutations in myeloid malignancies result in a dominant-negative effect without evidence of neomorphic gain-of-function activities, ultimately leading to a selection advantage when exposed to DNA damage.12 Thus, restoring wild-type function in TP53 mutant clones would be of profound beneficial impact

one is found in the bacterial cytoplasm (internal urease), and the other is present on the bacterial surface (external urease) (Sharndama and Mba 2022)

Wang J, Liu Y, Zhou LJ, Wu Y, Li F, Shen KF,

Psychiatry 71 DantzerR

Additionally, around 12% of oxygen used in mitochondrial metabolism produces free radicals, which can lead to oxidative stress and even DNA damage [2]