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STN stimulation and ECoG recording Before starting the clinical microelectrode recordings, sedation was temporarily suspended, ensuring that the patients were at their standard neurological status as determined by clinical evaluations

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Its downregulation has been shown to suppress the degradation of unsaturated fatty acids through multiple mechanisms, including inhibiting the activity of key enzymes involved in fatty acid catabolism (e.g., acetyl-CoA dehydrogenase, lipoxygenase, and fatty acid dioxygenase) and activating the PPAR signaling pathway and its downstream target genes [71]

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