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DK, SF, YL, XY and ZJ provided constructive suggestions for the article and contributed to its design and structuring

E.WallaceA.WoolleyJ

One is that the loss of a neuroprotective effect of UA when it is reduced below normal is not necessarily modelled by comparing serum levels in unaffected subjects with the hyperuricemia associated with gout

The production of thrombin is triggered by the exposure of tissue factors in the arterial wall, which in turn sets off the clotting cascade

The binding efficiency of these peptides was further validated using immunoprecipitation (IP) followed by immunoblotting (IB) in TLR5-overexpressing human intestinal epithelial (Caco-2) cells