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In addition to resistance to diet-induced obesity, the absence of HSL also protects against genetic obesity, based on the observation that mice homozygous for both HSL and leptin deficiency ( ob/ob ) are 26% lighter than ob/ob mice and this is reflected by a reduction in WAT mass [14]

AOD-9604 offers a gentler approach with more selective fat targeting and fewer systemic effects, making it suitable for individuals who cannot tolerate GLP-1 agonists or prefer a more focused mechanism