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The damage to HSCs that results in MYSM1 deficiencies is caused by ferroptosis

NQO1 metabolizes a two-electron reduction of -lapachone using NADH and NADPH as electron sources, forming an unstable hydroquinone that rapidly auto-oxidizes and generates ROS, thereby inducing cell death [136]

Heparin and vitamin K antagonists were the first non-specific medications used in anticoagulant therapy, followed by safer alternatives, such as fondaparinux, argatroban, and direct oral anticoagulants

Addressing nutritional gaps with multivitamin and mineral supplements

Molecular ion mass identification confirmed 1418 Da for BPC-157 and 4963 Da for TB-500