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lips-14 c elegans

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. Inactivation of SIAH-1 E3 ligase

Inactivation of SIAH 1 E3 ligase attenuates A toxicity by suppressing ubiquitin dependent DVE 1 degradation in Caenorhabditis elegans models of Alzheimer's disease Journal of Biological Chemistry Hermaphrodite Epithelial System Interfacial Cells WormAtlas C. elegans Introduction Handbook Epithelial System Hypodermis Handbook Nervous System Neuronal Support Cells

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Description

Empirical formula C Molar mass (M) 349,36 g/mol Storage temp

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. Inactivation of SIAH-1 E3 ligase

Spinal glial activation contributes to pathological pain states

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. Inactivation of SIAH-1 E3 ligase

It is designed to copy the structural and functional characteristics of Thymosin Beta-4

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. Inactivation of SIAH-1 E3 ligase

Stupp R, Taillibert S, Kanner A, Read W, Steinberg D, Lhermitte B, et al

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. Inactivation of SIAH-1 E3 ligase

K.PathakC

lips-14 c elegans Saturated very long chain fatty acid configures glycosphingolipid for lysosome homeostasis in long-lived C. Inactivation of SIAH-1 E3 ligase
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