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References 9295 show that copper chelators inhibit the mutant SOD toxicity, implying that copper has a pivotal role in the progression of ALS

Impaired mitochondrial biology may be subdivided into several levels, with several facets attributed to each [31]

Lack of oral GSH supplementation effects upon growth or changes in serum or fecal inflammatory markers in children with cystic fibrosis with pancreatic insufficiency (125) could be probably explained by the inability of the cells to uptake extracellular GSH to be used inside the cells

In cells, the presence of MTs confers resistance to the harmful effects of Cd, but the inability to produce MTs makes cells more susceptible to the metals effects (53)