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The epidermal lipid-microbiome loop and immunity: important players in atopic dermatitis

Accumulating evidence suggests that metabolic disturbances can promote the development of AF through structural and electrophysiological remodelling, but the underlying mechanisms that predispose an individual to AF are aetiology-dependent, thus emphasizing the need for tailored therapeutic strategies to treat AF that target an individuals metabolic profile

Roth S, Spalinger MR, Gottier C, Biedermann L, Zeitz J, Lang S

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Concurrently, to reverse neurological damage, innovative strategies should be explored including glial cell reprogramming (e.g., tamoxifen derivatives inducing neuroprotective astrocytes) and microbial-mitochondrial axis interventions, combined with high-throughput screening of BBB-protective agents using organoid models