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In this context, SHMT2 initially functions as a scaffold protein that recruits BRISC to ubiquitinated IFNAR1, promoting K63-linked deubiquitination and preventing receptor degradation to sustain type I interferon signaling [130]

These receptors are GIP (Glucose Inhibitory Peptide or Glucose-Dependent Insulinotropic Peptide) and GLP (Glucagon-Like Peptide-1)

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Michaowicz J (2014) Bisphenol A - sources, toxicity and biotransformation

As the NAC regimen (with an iv -priming dose) was more intense in severe than moderate IPTB cases, it is unclear whether differences are due to disease severity or to NAC treatment per se