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Its probable mechanism includes activation of renal AGE-RAGE signaling pathways, decreasing levels of monocyte chemotactic protein-1 (MCP-1) and PKC-A, which consequently reduces the production of slit diaphragm proteins such as nephrin and podocin, thereby mitigating the impact of AGE-mediated DN pathogenesis (Muthenna et al., 2014)

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308 , F1259F1267 (2015)

conducted a double-blind, placebo-controlled randomized controlled trial to examine the effect of a GlyNAC supplementation on GSH concentrations, oxidative stress (OxS), mitochondrial fatty-acid oxidation (MFO), insulin resistance (IR), inflammation, endothelial function, physical function, and body composition