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Therefore, accurate calculations of the anion gap were not possible for all patients with acidosis

Moreover, ferroptosis in this cell line can be assessed using the classic cytotoxic lactate dehydrogenase (LDH) assay (see figure)

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This suggests that NOX2-dependent inflammatory re-routing plays a significant role in the development and maintenance of exacerbated SLE in people whose NOX2 complex activity is low ( 2.3 Malfunction of NCF1 and NOX2 in SLE Patients with SLE lack phagocytic oxidative burst because they have mutations in essential phagocyte NOX2 complex subunits