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The objective of the present study was to determine the effect of chronic exposure to 3MC, a major PAH, on the transsulfuration pathway using targeted metabolomics analysis, aiming to provide novel insights into the mechanism through which 3MC affects intracellular GSH levels and renders cells vulnerable to a second insult of oxidative stress

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T 4 , thyroxine (3,5,3,5-tetraiodothyronine), is produced by follicular cells of the thyroid gland

Heme oxygenase 1 (HMOX-1/HO-1), a well-known antioxidant enzyme, initiates heme degradation to release iron [27]