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J Nutr Biochem 70:215226

HMGB1 deficiency reduces H 2 O 2 -induced oxidative damage in human melanocytes via the Nrf2 pathway

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In addition to directly targeting enzymes related to glucose metabolism, it is also feasible to target common factors influencing glucose metabolism, such as HIF-1

These behavioral benefits are underpinned by its marked ability to induce spinogenesis and promote functional synaptogenesis in hippocampal neurons, highlighting its potential as a robust procognitive and antidementia agent [2]